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Symbian does not provide for automatic protections against integer overflows An integer overflow occurs when the memory representation of an integer variable lacks the capacity to hold the result of an arithmetic operation The behavior when such a condition arises is left as an implementation-specific detail Integer overflows can cause security vulnerabilities in many instances for example, when allocating memory or accessing an array Developers must consciously take strides to eliminate the risk associated with such errors As such, each arithmetic operation should be checked for overflow conditions When a type is available whose representation is twice as large as the type that is to be checked for example, a 64-bit integer and a 32-bit integer the overflow tests are straightforward This is because the larger type can accurately hold the result of any arithmetic operation on variables of the smaller type Symbian provides both a signed and an unsigned 64-bit integer type that can be used to perform such testing:

Onset is usually within the rst 3 days of life The most notable features include a weak cry, dif culty feeding due to a poor suck, hypotonia, respiratory dif culty, ptosis, and diminished facial expression (facial muscle weakness) The disorder is temporary, with a mean duration of about 18 20 days The most important aspect of this disorder is that it is only temporary and there appears to be no increased risk of the child developing MG in later life

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Effective management of MG requires knowledge of neoplastic and other autoimmune disorders that occur with an increased incidence in patients with MG It is tempting to speculate that many of these historically described cases were examples of Dok-7 myasthenia described previously Pathology of the thymus gland is the most notorious disease association132 As many as 70% of patients with MG have thymic hyperplasia, while approximately 10% have a thymoma133,134 Conversely, 55% of patients with thymoma will have MG71 Thymomas are much more common in patients between the ages of 50 and 70 years Thymomas have been reported to be associated with other neurological and neuromuscular disorders as well, including granulomatous myositis, Issac s syndrome, rippling muscle disease limbic and cerebellar encephalitis, and autonomic neuropathy including the syndrome of intestinal pseudoobstruction70,71,135,136 Seventy-four percent of patients with thymomas and myasthenia have circulating serum striational antibodies in addition to anti-AChR antibodies71 Less than half that number with thymoma will have striational antibodies without MG Other autoantibodies can be found in patients with thymoma, with or without MG, including ganglionic (as opposed to the nicotinic found on muscle) AChR antibodies, anti-voltage-gated potassium channel antibodies, and CRMP-5-IgG70,71 Thymic abnormalities in patients with anti-MuSK antibodies are uncommon and typically minimal when they occur88 This point is emphasized in a study of 167 patients with thymoma71 Of the 92 who had MG, only one of them was seropositive for MuSK antibodies, which rarely, if ever, coexist with their AChR-binding Other autoimmune diseases that occur with increased frequency in a myasthenic population (and vice versa) include rheumatoid arthritis, systemic lupus erythematosus, Sj gren syndrome, red blood cell aplasia, o ulcerative colitis, sarcoid, Addison disease, and hyperand hypothyroidism31,137 140 Other neuromuscular disorders can be seen concurrently in patients with MG as well, particularly if thymoma is coexistent141 Both acute and chronic in ammatory demyelinating polyneuropathies have been reported in patients with concurrent MG142 144 There have been reports of patients who develop a severe.

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Sural nerve biopsies reveal a loss of large and small myelinated nerve bers with relative preservation of unmyelinated axons Remaining axons are thinly myelinated, and onion-bulb formations may also be evident

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of conduction between different nerves and along different segments of individual nerves resulting in temporal dispersion has been described in some cases of CMT1X, particularly in women176,177 Motor unit nerve estimates demonstrate a reduction in units, which correlates with clinical severity170

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Sensory nerve biopsies reveal a loss of myelinated nerve bers, especially of large-diameter bers, along with axonal degeneration and atrophy, and clusters of thinly myelinated regenerating bers10,167,174,176 A mild degree of Schwann cell proliferation (onion bulbs) can also be seen surrounding some of the thinly myelinated bers A mixture of demyelination and remyelination is evident on teased ber preparations

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